index - Organisation de la cellule musculaire et thérapie de la myopathie centronucléaire autosomique dominante Accéder directement au contenu

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Cancer Atrial heart defects AD-CNM Centronuclear myopathy Cross-presentation Adeno-Associated virus BAR proteins Dynamin 2 Nesprin Ctdnep1 Endocytosis Cavéoles Dystrophie musculaire d'Emery Dreifuss Cardiomyopathies BAF AAV8 Duchenne Muscular Dystrophy Charcot-Marie-Tooth Actin nucleus Coeur Gene therapy AAV Caveolin Dynamin Adhesion Cellular neuroscience Allele‐specific silencing therapy Lamin Cell migration Cytoskeleton A-type lamins CAV-3 gene Allele-specific silencing Autophagosome Myosin Developmental myosin heavy chain Mechanotransduction Duchenne muscular dystrophy Caveolins CTL Biomarkers Skeletal muscle Dominant centronuclear myopathy Nuclear envelope Muscular dystrophy Allele specific RNA interference Autophagy cellular Correlative microscopy Becker muscular dystrophy BMD Satellite cell Cell proliferation Adeno-associated virus vector Dynamin overexpression Cross-bridge kinetics ACTN2 Dynamine Myopathy Alpha-actinin-2 BMP signaling Dullard Adeno-associated virus Autophagosome maturation Actin Clathrine Autosomal dominant centronuclear myopathy Disease modifiers Migration Skin Myopathie Cardiotoxin Animal models of human disease Allele-specific silencing therapy Cell signaling Biophysics Antisense oligonucleotides Clathrin Atrial cardiac defects Diaphragm Autophagy Cavins DMyHC Cellules de crête neurale RNA interference Caveolae Developmental biology Nucleus Disease heterogeneity Core myopathy Duchenne muscular dystrophy DMD Muscle Outflow tract Amphiphysin Neural crest cells Domaine LEM Congenital myopathy AFM Dystrophie musculaire de Duchenne Cytosquelette Adult patients DNM2